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CYP2A6 is the phase I regulator of a great amount of endogenous and exogenous molecules [75], and its elevation is related to the development of liver pathologies [76]

In view of the new technologies in the investigation and development of pharmacology, it is necessary to advance in the therapeutics of these patients with fatty liver, mainly without diabetes, due to that, according to Cho et al

The p65 subunit of NF-B inhibits the Nrf2/ARE pathway by depriving CREB binding protein (CBP), allowing HDAC3 to recruit to MafK and interact with Keap1 (262, 263)